@article{475d03c64a4a44c59aabaf515a350b61,
title = "Loss of CRMP2 O-GlcNAcylation leads to reduced novel object recognition performance in mice",
keywords = "Cognitive function, CRMP2, Crosstalk, O-GlcNAcylation, collapsin response mediator protein-2, n acetylglucosamine, nerve protein, signal peptide, aging, amino acid sequence, animal, C57BL mouse, cell line, chemistry, cognition, exploratory behavior, female, gene knock-in, genetics, human, male, memory disorder, metabolism, mouse, point mutation, protein processing, short term memory, Acetylglucosamine, Aging, Amino Acid Sequence, Animals, Cell Line, Cognition, Exploratory Behavior, Female, Gene Knock-In Techniques, Humans, Intercellular Signaling Peptides and Proteins, Male, Memory Disorders, Memory, Short-Term, Mice, Mice, Inbred C57BL, Nerve Tissue Proteins, Point Mutation, Protein Processing, Post-Translational, 1182 Biochemistry, cell and molecular biology",
author = "Villo Muha and Ritchie Williamson and Rachel Hills and A.D. McNeilly and T.G. McWilliams and Jana Alonso and Marianne Schimpl and Leney, \{Aneika C.\} and Heck, \{Albert J.R.\} and Calum Sutherland and Read, \{Kevin D.\} and McCrimmon, \{Rory J.\} and S.P. Brooks and \{Van Aalten\}, \{Daan M.F.\}",
year = "2019",
doi = "10.1098/rsob.190192",
language = "English",
volume = "9",
journal = "Open biology",
issn = "2046-2441",
publisher = "Royal Society Publishing",
number = "11",
}