Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL

A. Schrader, G. Crispatzu, S. Oberbeck, P. Mayer, S. Pützer, J. von Jan, E. Vasyutina, K. Warner, N. Weit, N. Pflug, T. Braun, E. I. Andersson, B. Yadav, A. Riabinska, B. Maurer, M. S. Ventura Ferreira, F. Beier, J. Altmueller, M. Lanasa, C. D. Herling & 16 andra T. Haferlach, S. Stilgenbauer, G. Hopfinger, M. Peifer, T. H. Brümmendorf, P. Nürnberg, K. S. J. Elenitoba-Johnson, S. Zha, M. Hallek, R. Moriggl, H. C. Reinhardt, M. -H. Stern, S. Mustjoki, S. Newrzela, P. Frommolt, M. Herling

Forskningsoutput: Tidskriftsbidrag › Artikel › Vetenskaplig › Peer review

Originalspråkengelska
Artikelnummer697
TidskriftNature Communications
Volym9
Antal sidor22
ISSN2041-1723
DOI
StatusPublicerad - 15 feb 2018
MoE-publikationstypA1 Tidskriftsartikel-refererad

Vetenskapsgrenar

  • 3111 Biomedicinska vetenskaper
  • 3122 Cancersjukdomar

Citera det här

Schrader, A., Crispatzu, G., Oberbeck, S., Mayer, P., Pützer, S., von Jan, J., ... Herling, M. (2018). Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL. Nature Communications, 9, [697]. https://doi.org/10.1038/s41467-017-02688-6
Schrader, A. ; Crispatzu, G. ; Oberbeck, S. ; Mayer, P. ; Pützer, S. ; von Jan, J. ; Vasyutina, E. ; Warner, K. ; Weit, N. ; Pflug, N. ; Braun, T. ; Andersson, E. I. ; Yadav, B. ; Riabinska, A. ; Maurer, B. ; Ferreira, M. S. Ventura ; Beier, F. ; Altmueller, J. ; Lanasa, M. ; Herling, C. D. ; Haferlach, T. ; Stilgenbauer, S. ; Hopfinger, G. ; Peifer, M. ; Brümmendorf, T. H. ; Nürnberg, P. ; Elenitoba-Johnson, K. S. J. ; Zha, S. ; Hallek, M. ; Moriggl, R. ; Reinhardt, H. C. ; Stern, M. -H. ; Mustjoki, S. ; Newrzela, S. ; Frommolt, P. ; Herling, M. / Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL. I: Nature Communications. 2018 ; Vol. 9.
@article{bdb50aa8009e404d87f4bf531282968e,
title = "Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL",
keywords = "CELL PROLYMPHOCYTIC LEUKEMIA, CHRONIC LYMPHOCYTIC-LEUKEMIA, ACUTE MYELOID-LEUKEMIA, DOUBLE-STRAND BREAK, ATM KINASE-ACTIVITY, ATAXIA-TELANGIECTASIA, DNA-DAMAGE, GENOMIC INSTABILITY, EMBRYONIC LETHALITY, TELOMERASE ACTIVITY, 3111 Biomedicine, 3122 Cancers",
author = "A. Schrader and G. Crispatzu and S. Oberbeck and P. Mayer and S. P{\"u}tzer and {von Jan}, J. and E. Vasyutina and K. Warner and N. Weit and N. Pflug and T. Braun and Andersson, {E. I.} and B. Yadav and A. Riabinska and B. Maurer and Ferreira, {M. S. Ventura} and F. Beier and J. Altmueller and M. Lanasa and Herling, {C. D.} and T. Haferlach and S. Stilgenbauer and G. Hopfinger and M. Peifer and Br{\"u}mmendorf, {T. H.} and P. N{\"u}rnberg and Elenitoba-Johnson, {K. S. J.} and S. Zha and M. Hallek and R. Moriggl and Reinhardt, {H. C.} and Stern, {M. -H.} and S. Mustjoki and S. Newrzela and P. Frommolt and M. Herling",
year = "2018",
month = "2",
day = "15",
doi = "10.1038/s41467-017-02688-6",
language = "English",
volume = "9",
journal = "Nature Communications",
issn = "2041-1723",
publisher = "Nature Publishing Group",

}

Schrader, A, Crispatzu, G, Oberbeck, S, Mayer, P, Pützer, S, von Jan, J, Vasyutina, E, Warner, K, Weit, N, Pflug, N, Braun, T, Andersson, EI, Yadav, B, Riabinska, A, Maurer, B, Ferreira, MSV, Beier, F, Altmueller, J, Lanasa, M, Herling, CD, Haferlach, T, Stilgenbauer, S, Hopfinger, G, Peifer, M, Brümmendorf, TH, Nürnberg, P, Elenitoba-Johnson, KSJ, Zha, S, Hallek, M, Moriggl, R, Reinhardt, HC, Stern, M-H, Mustjoki, S, Newrzela, S, Frommolt, P & Herling, M 2018, 'Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL', Nature Communications, vol. 9, 697. https://doi.org/10.1038/s41467-017-02688-6

Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL. / Schrader, A.; Crispatzu, G.; Oberbeck, S.; Mayer, P.; Pützer, S.; von Jan, J.; Vasyutina, E.; Warner, K.; Weit, N.; Pflug, N.; Braun, T.; Andersson, E. I.; Yadav, B.; Riabinska, A.; Maurer, B.; Ferreira, M. S. Ventura; Beier, F.; Altmueller, J.; Lanasa, M.; Herling, C. D.; Haferlach, T.; Stilgenbauer, S.; Hopfinger, G.; Peifer, M.; Brümmendorf, T. H.; Nürnberg, P.; Elenitoba-Johnson, K. S. J.; Zha, S.; Hallek, M.; Moriggl, R.; Reinhardt, H. C.; Stern, M. -H.; Mustjoki, S.; Newrzela, S.; Frommolt, P.; Herling, M.

I: Nature Communications, Vol. 9, 697, 15.02.2018.

Forskningsoutput: Tidskriftsbidrag › Artikel › Vetenskaplig › Peer review

TY - JOUR

T1 - Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL

AU - Schrader, A.

AU - Crispatzu, G.

AU - Oberbeck, S.

AU - Mayer, P.

AU - Pützer, S.

AU - von Jan, J.

AU - Vasyutina, E.

AU - Warner, K.

AU - Weit, N.

AU - Pflug, N.

AU - Braun, T.

AU - Andersson, E. I.

AU - Yadav, B.

AU - Riabinska, A.

AU - Maurer, B.

AU - Ferreira, M. S. Ventura

AU - Beier, F.

AU - Altmueller, J.

AU - Lanasa, M.

AU - Herling, C. D.

AU - Haferlach, T.

AU - Stilgenbauer, S.

AU - Hopfinger, G.

AU - Peifer, M.

AU - Brümmendorf, T. H.

AU - Nürnberg, P.

AU - Elenitoba-Johnson, K. S. J.

AU - Zha, S.

AU - Hallek, M.

AU - Moriggl, R.

AU - Reinhardt, H. C.

AU - Stern, M. -H.

AU - Mustjoki, S.

AU - Newrzela, S.

AU - Frommolt, P.

AU - Herling, M.

PY - 2018/2/15

Y1 - 2018/2/15

KW - CELL PROLYMPHOCYTIC LEUKEMIA

KW - CHRONIC LYMPHOCYTIC-LEUKEMIA

KW - ACUTE MYELOID-LEUKEMIA

KW - DOUBLE-STRAND BREAK

KW - ATM KINASE-ACTIVITY

KW - ATAXIA-TELANGIECTASIA

KW - DNA-DAMAGE

KW - GENOMIC INSTABILITY

KW - EMBRYONIC LETHALITY

KW - TELOMERASE ACTIVITY

KW - 3111 Biomedicine

KW - 3122 Cancers

U2 - 10.1038/s41467-017-02688-6

DO - 10.1038/s41467-017-02688-6

M3 - Article

VL - 9

JO - Nature Communications

JF - Nature Communications

SN - 2041-1723

M1 - 697

ER -

Schrader A, Crispatzu G, Oberbeck S, Mayer P, Pützer S, von Jan J et al. Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL. Nature Communications. 2018 feb 15;9. 697. https://doi.org/10.1038/s41467-017-02688-6

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